Myasthenia gravis (my-us-THEE-nee-uh GRAY-vis) is characterized by weakness and rapid fatigue in one of the muscles that you voluntarily control. It is caused by a disruption in normal communication between nerves and muscles.
There is no cure for myasthenia gravis, but treatment can help relieve the signs and symptoms, such as: B. Weakness in the muscles of the arms or legs, double vision, droopy eyelids, and difficulty walking. Talking, chewing, swallowing, and breathing.
Although this disease can affect people of any age, it is more common in women under 40 and in men over 60. Myasthenia gravis Treatment in Khammam
The muscle weakness caused by myasthenia gravis worsens when the affected muscle is used. Since symptoms usually improve with rest, muscle weakness can come and go. However, symptoms continue to develop over time and usually reach their worst state within a few years of onset.
Although myasthenia gravis can affect all muscles that you voluntarily control, some muscle groups are affected more often than others.
Receptors for neurotransmitters
ReceiversOpen Pop-up Dialog
Your nerves communicate with your muscles by releasing chemicals (neurotransmitters) that fit precisely into muscle cell receptor sites on the nerve-muscle junction.
- In myasthenia gravis, your immune system makes antibodies that block or destroy many receptor sites in your muscles for a neurotransmitter called acetylcholine (as-uh-teel-KOH-leen). When fewer receptor sites are available, your muscles receive fewer nerve signals, which leads to weakness.
- Antibodies can also block the function of a protein called muscle-specific receptor tyrosine kinase (TIE-roh-vu KIE-nays). This protein is involved in the formation of the nerve-muscle junction. Antibodies that block this protein can lead to myasthenia gravis. Myasthenia gravis Treatment in Khammam